🧐Why People with Thyroid Disease Are at Increased Risk of Heart Disease Thyroid-Dr Richard Hagmeyer

Описание: In This Video: 🧐Why People with Thyroid Disease Are at Increased Risk of Heart Disease Thyroid-Dr Richard Hagmeyer


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Why People with Thyroid Disease Are at Increased Risk of Heart Disease – Dangers of High Homocysteine

In this video, Dr. Richard Hagmeyer explains the connection between thyroid disease and heart disease, focusing on the dangers of high homocysteine levels. People with thyroid disorders, such as hypothyroidism or hyperthyroidism, are often at an increased risk of cardiovascular problems, including heart attacks and strokes.

Discover how high homocysteine, a common but overlooked risk factor, contributes to heart disease in thyroid patients. Dr. Hagmeyer also shares tips on how to manage homocysteine levels naturally, as well as the critical lifestyle changes and medical strategies you can implement to protect your heart health if you have a thyroid condition.

Key Points Covered:

How thyroid dysfunction affects heart health
The role of homocysteine in cardiovascular disease
Steps to lower homocysteine levels
Importance of diet, lifestyle, and supplements in managing heart disease risk

Stay informed and take proactive steps to manage your thyroid and heart health!

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In today’s video, I want to explain

• The connection between MTHFR and Thyroid disease and Hashimotos
• The problem with high Homocysteine
• 7 essential nutrients needed for proper methylation and MTHFR function
• What you need to do from a lifestyle perspective if you have an MTHFR variant.

Thyroid disease and Hashimotos are complex health problems with many layers and the genetic information that can be obtained by testing for the MTHFR variant can help your doctor when it comes to treatment.
Our bodies are bombarded day in and day out with toxins and a variety of pollutants.
Throw in poor dietary choices, daily medications, stress, hormone imbalances, gut problems, and gluten sensitivity and we really impact a process known as methylation.


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